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1.
FIG. 1.

FIG. 1. From: Outer Membrane Protein Expression Profile in Helicobacter pylori Clinical Isolates .

OMP expression profiles of representative H. pylori isolates. Whole-cell lysates of H. pylori strains were separated in a 10% SDS-polyacrylamide gel (or a 6% gel for CagA detection), blotted on a nitrocellulose membrane, and detected with antisera against eight different OMPs or CagA.

Stefan Odenbreit, et al. Infect Immun. 2009 Sep;77(9):3782-3790.
2.
FIG. 5.

FIG. 5. From: Outer Membrane Protein Expression Profile in Helicobacter pylori Clinical Isolates .

Graphic representation of omp gene expression patterns of 200 H. pylori isolates by use of a heat map. Two hundred H. pylori isolates are shown in a two-dimensional representation in which the presence of a certain OMP (AlpA, AlpB, HP0227, OipA, BabA, BabB, HP0317, SabA, and CagA) is indicated by a gray line and its absence is shown by a white line. The relatedness of certain isolates concerning a common expression pattern is shown in one dimension (rows), and the coexpression patterns of all omp genes are shown in the other dimension (columns).

Stefan Odenbreit, et al. Infect Immun. 2009 Sep;77(9):3782-3790.
3.
FIG. 4.

FIG. 4. From: Outer Membrane Protein Expression Profile in Helicobacter pylori Clinical Isolates .

IL-8 production from AGS cells. AGS cells were infected with representative H. pylori strains classified according to CagA and OipA production (MOI = 100; infection was done for 4 hours). The IL-8 concentration in the supernatant was measured by ELISA. Each value was calculated as a ratio to the amount of IL-8 produced by the IL-8-inducing wt strain 26695, which was set to 100%. The line in each bar represents the 50th percentile (median) of the group; the ends of the bars indicate the 25th and 75th percentiles. Error bars show the 10th and 90th percentiles, and single dots include 5th and 95th percentile outliers.

Stefan Odenbreit, et al. Infect Immun. 2009 Sep;77(9):3782-3790.
4.
FIG. 2.

FIG. 2. From: Outer Membrane Protein Expression Profile in Helicobacter pylori Clinical Isolates .

Binding of H. pylori control strains to defined receptor structures. (A) Strains were classified according to the production of BabA and SabA proteins. (B) Production of BabA and SabA in well-characterized control strains. (C) Binding of fluorescently labeled H. pylori bacteria of these strains to defined antigens, including Leb-HSA, laminin, collagen type IV, and fibronectin, was explored by dot blot overlay assays. J99 and CCUG17874 are wt strains, and J99(babA) and J99(sabA) are isogenic mutant strains with chromosomal deletions of the genes in parentheses (, ).

Stefan Odenbreit, et al. Infect Immun. 2009 Sep;77(9):3782-3790.
5.
FIG. 3.

FIG. 3. From: Outer Membrane Protein Expression Profile in Helicobacter pylori Clinical Isolates .

Binding of H. pylori clinical isolates to defined receptor structures. (A) Binding of fluorescently labeled H. pylori (Hp) bacteria of six representative isolates to defined antigens, including Leb-HSA, laminin, fibronectin, and collagen type IV, was explored by dot blot overlay assays. Production of BabA, SabA, and CagA is indicated below. (B) Characterization of some strains expressing inactive BabA proteins. No binding to Leb-HSA or the derivatives ALeb-HSA and BLeb-HSA could be observed. (C) Production of “inactive” SabA proteins in two H. pylori isolates.

Stefan Odenbreit, et al. Infect Immun. 2009 Sep;77(9):3782-3790.

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